Rosen & Barkin's 5-Minute Emergency Medicine Consult (119 page)

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Authors: Jeffrey J. Schaider,Adam Z. Barkin,Roger M. Barkin,Philip Shayne,Richard E. Wolfe,Stephen R. Hayden,Peter Rosen

Tags: #Medical, #Emergency Medicine

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FOLLOW-UP
DISPOSITION
Admission Criteria
  • Injuries requiring admission
    • Partial-thickness burns of noncritical areas (excludes eyes, ears, face, hands, feet, or perineum) involving 10–20% of BSA in adults (>10 yr and <50 yr)
    • Partial-thickness burns of noncritical areas involving 5–10% of BSA in children <10 yr
    • Suspicion of nonaccidental trauma
    • Patients unable to care for wounds in outpatient setting (e.g., homeless patients)
  • Injuries requiring transfer/admission to a burn center
    • Partial-thickness or full-thickness burns involving ≥10% of BSA
    • Full-thickness burns involving >5% of BSA
    • Partial-thickness and full-thickness of face, hands, feet, genitalia, perineum, or major joints
    • Electrical burns, including lightning injury
    • Significant chemical burns
    • Inhalation injury
    • Patients with pre-existing illness that could complicate management
    • Patients with concomitant trauma or social barriers
Discharge Criteria

Partial-thickness burns of <10% of BSA in adults (<5% in children or the elderly) involving noncritical areas only. Patients must be reliable, able to manage wounds as outpatients and obtain follow-up.

Issues for Referral

Maintain low threshold for referral to burn specialist whenever there is raised concern regarding cosmesis, involvement of hands/face/eyes/perineum, or if burn is overlying a joint.

FOLLOW-UP RECOMMENDATIONS

1–2 days after the injury to assess for early infection, saturation of dressings, pain control

PEARLS AND PITFALLS
  • Early IV fluid rehydration is essential
  • Intubate early for signs of respiratory distress; must recognize potential for airway involvement
  • Early pain control in all burns
  • Monitor for hypoglycemia in children
ADDITIONAL READING
  • Committee on Trauma, American College of Surgeons. Guidelines for the operation of burn units.
    Resources for Optimal Care of the Injured Patient
    ; 2006:79–86.
  • Pham TN, Cancio LC, Gibran NS. American Burn Association practice guidelines burn shock resuscitation.
    J Burn Care Res
    . 2008;29(1):257–266.
  • Toon MH, Maybauer MO, Greenwood JE, et al. Management of acute smoke inhalation injury.
    Crit Care Resusc.
    2010;12:53–61.
  • Cancio LC, Lundy JB, Sheridan RL. Evolving changes in the management of burns and environmental injuries.
    Surg Clin North Am
    . 2012;92(4):959–986.
CODES
ICD9
  • 949.0 Burn of unspecified site, unspecified degree
  • 949.1 Erythema [first degree], unspecified site
  • 949.2 Blisters, epidermal loss [second degree], unspecified site
ICD10
  • T20.00XA Burn of unsp degree of head, face, and neck, unsp site, init
  • T30.0 Burn of unspecified body region, unspecified degree
  • T30.4 Corrosion of unspecified body region, unspecified degree
BURSITIS
Patrick H. Sweet
BASICS
DESCRIPTION
  • Bursae are synovial fluid-filled sacs:
    • ∼150 are located between bones, ligaments, tendons, muscles, and skin.
  • They provide lubrication to reduce friction during movement.
  • Bursitis is inflammation of a bursa caused by trauma and overuse, infection, crystal deposition, or systemic disease.
  • Chronic bursitis can lead to proliferative changes in the bursa.
  • Commonly affected sites:
    • Shoulder (subacromial)
    • Elbow (olecranon): Usually secondary to trauma
    • Hip (greater trochanter, ischial, iliopsoas): More common in elderly
    • Knee (prepatellar and pes anserine): Secondary to trauma or arthritis
    • Foot (calcaneal): Almost always secondary to improperly fitting shoes/heels
ETIOLOGY
  • Trauma (most common cause):
    • Specific traumatic event or repetitive use of associated joints
  • Infection: Secondary to direct penetration; may be obvious or microscopic:
    • Higher risk with diabetes, chronic alcohol abuse, uremia, gout, immunosuppression
    • 90% caused by
      Staphylococcus
      spp.
  • Crystal deposition: Calcium phosphate, urate
  • Systemic disease: Rheumatoid arthritis, gout, ankylosing spondylitis, psoriatic arthritis, lupus, rheumatic fever
DIAGNOSIS
SIGNS AND SYMPTOMS
History
  • Acute or chronic
  • History of trauma, overuse, or prolonged pressure
  • Pain with increased activity at respective joint or with pressure
  • Functional complaints (e.g., limping)
  • History of localized swelling
  • Screen for symptoms of systemic disease
  • History of gout or pseudogout or rheumatologic disease
  • History of recent procedure at bursa (e.g., aspiration, injection, etc.)
Physical-Exam
  • Tenderness to palpation is minimal to mild in aseptic bursitis.
  • Localized pain with movement
  • Often reduced active range of motion (ROM) with preserved passive ROM
  • Localized swelling, particularly with superficial bursae
  • Skin trauma overlying bursa
  • Warmth and erythema*
  • May be febrile in septic bursitis

*NB: The constellation of erythema, warmth, swelling, and exquisite tenderness are common in septic bursitis.

ESSENTIAL WORKUP
  • Full assessment of adjacent musculoskeletal structures
  • Any suspicion of infection warrants aspiration of bursae (especially olecranon and prepatellar bursae).
  • Lateral approach to prevent a needle tract directly over lines of tension of the joint
  • Aspiration of hip and other deep bursae may be guided in ED by US or deferred to consultants.
DIAGNOSIS TESTS & NTERPRETATION
Lab
  • Serum labs:
    • Suspected infection: CBC with differential
    • Evaluation of related systemic disease (e.g., uric acid level for gout); ESR and CRP in rheumatologic disease
    • Send serum glucose if bursal fluid aspiration is done
  • Bursal fluid analysis:
    • Send fluid for complete cell count with differential, glucose, total protein, crystal determination, Gram stain, and culture.
    • Cultures must always be sent.
    • Normal fluid: Fluid is clear yellow with 0–200 WBCs, 0 RBCs, low protein, and glucose is same as serum.
    • Traumatic bursitis: Fluid is bloody/xanthochromic with <1,200 WBCs, many RBCs, low protein, and normal glucose.
    • Septic bursitis: Fluid is cloudy yellow with >50,000 WBCs, few RBCs, slightly increased protein, and decreased glucose; bacteria on Gram stain.
    • Rheumatoid and microcrystalline inflammation (aseptic bursitis): Fluid is yellow, sometimes turbid, and has 1,000–40,000 WBCs, few RBCs, slightly increased protein, and variable glucose; microscopic exam for crystals.
Imaging
  • Radiographs may demonstrate soft tissue swelling or adjacent chronic arthritic changes or calcium deposits:
    • Especially recommended when trauma is involved to rule out fracture or foreign body
  • MRI and US may aid in diagnosis of deep bursitis and in defining the extent of septic bursitis.
  • CT scans can also help differentiate septic from nonseptic bursitis.
DIFFERENTIAL DIAGNOSIS
  • Arthritides: Septic, inflammatory, rheumatoid and degenerative joint (osteoarthritis)
  • Gout and pseudogout
  • Tendonitis, fasciitis, epicondylitis
  • Fracture, tendon/ligament tear, contusion, sprain
  • Osteomyelitis
  • Nerve entrapment
  • Also in hips: Neuritis, lumbar spine disease, sacroiliitis
TREATMENT
PRE HOSPITAL

May be difficult to distinguish from fractures; suspicious joints should be immobilized, particularly in the setting of trauma.

INITIAL STABILIZATION/THERAPY
  • Immobilize joint if pain is severe.
  • Shoulders should not be immobilized for >2–3 days because of the risk of adhesive capsulitis.

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