Authors: Mary A. Williamson Mt(ascp) Phd,L. Michael Snyder Md
Phosphate enemas, laxatives or infusions
Excess vitamin D intake
IV therapy for hypophosphatemia or hypercalcemia
Milk-alkali (Burnett) syndrome (some patients)
Massive blood transfusions
Hemolysis of blood
Miscellaneous
High intestinal obstruction
Sarcoidosis (some patients)
Decreased In
Primary hypophosphatemia
Decreased GI absorption
Decreased dietary intake
Decreased intestinal absorption, for example, malabsorption, steatorrhea, secretory diarrhea, vomiting, vitamin D deficiency, drugs (antacids, alcohol, glucocorticoids)
Decreased renal tubular reabsorption (>100 mg/day in urine during hypophosphatemia indicates excessive renal loss)